The Lancet JUNE 09, 2018

Dabigatran in patients with myocardial injury after non-cardiac surgery (MANAGE): an international, randomised, placebo-controlled trial

Devereaux PJ, Duceppe E, Guyatt G, et al., on behalf of the MANAGE Investigators

Bottom Line

In patients with myocardial injury after non-cardiac surgery (MINS), treatment with dabigatran 110 mg twice daily significantly reduced the risk of major vascular complications without a significant increase in major bleeding compared with placebo.

Key Findings

1. Dabigatran 110 mg twice daily reduced the primary composite outcome of major vascular complications (vascular mortality, non-fatal myocardial infarction, non-hemorrhagic stroke, peripheral arterial thrombosis, amputation, or symptomatic venous thromboembolism) compared with placebo (11% vs. 15%; hazard ratio 0.72; 95% CI 0.55–0.93; p=0.0115).
2. There was no statistically significant increase in the risk of major, life-threatening, or critical organ bleeding between the dabigatran and placebo groups (3% vs. 4%; hazard ratio 0.92; 95% CI 0.55–1.53; p=0.76).
3. The study was terminated early due to loss of funding and slow recruitment, which potentially limits the power and generalizability of the findings.
4. A high rate of study drug discontinuation was observed, occurring in 46% of patients in the dabigatran arm and 43% in the placebo arm, which complicates adherence and long-term interpretation.

Study Design

Design
RCT
Double-Blind
Sample
1,754
Patients
Duration
16 mo
Median
Setting
Multicenter, international
Population Patients aged 45 years or older who had undergone non-cardiac surgery and were within 35 days of suffering myocardial injury after non-cardiac surgery (MINS).
Intervention Dabigatran 110 mg orally twice daily.
Comparator Matched placebo.
Outcome Composite of major vascular complications, including vascular mortality, non-fatal myocardial infarction, non-hemorrhagic stroke, peripheral arterial thrombosis, amputation, and symptomatic venous thromboembolism.

Study Limitations

The trial was terminated prematurely, reducing the planned sample size and potentially affecting the robustness of the primary endpoint analysis.
The primary outcome definition was modified during the conduct of the trial.
High rates of treatment discontinuation (over 40% in both groups) reflect real-world challenges in maintaining long-term postoperative anticoagulation.
The majority of enrolled patients (80%) had isolated, asymptomatic troponin elevation, which may not be fully representative of patients with overt clinical myocardial infarction.

Clinical Significance

The MANAGE trial provides the first randomized evidence suggesting that postoperative anticoagulation with dabigatran may mitigate the high cardiovascular risk associated with MINS, an often-overlooked entity that independently predicts short- and long-term mortality.

Historical Context

MINS was historically under-recognized due to its frequent lack of ischemic symptoms. Previous research identified MINS as a major independent predictor of 30-day and 1-year mortality, yet clinical practice lacked established, evidence-based management strategies until this trial evaluated the role of targeted anticoagulation.

Guided Discussion

High-yield insights from every perspective

Med Student
Medical Student

What is the primary pathophysiology of Myocardial Injury after Non-cardiac Surgery (MINS), and how does the mechanism of dabigatran address the subsequent risk of vascular events?

Key Response

MINS is often asymptomatic and detected only by troponin elevation; it involves both Type 1 MI (plaque rupture) and Type 2 MI (supply-demand mismatch). Dabigatran is a direct thrombin inhibitor that prevents the conversion of fibrinogen to fibrin and inhibits thrombin-induced platelet aggregation, thereby reducing the formation of thrombi in the high-stress, prothrombotic state that follows major surgery.

Resident
Resident

In a patient diagnosed with MINS who is already on Aspirin for secondary prevention, how should the findings of the MANAGE trial influence your decision to add dabigatran 110 mg twice daily, and what clinical monitoring is essential?

Key Response

The MANAGE trial demonstrated that dabigatran reduced a composite of major vascular complications (HR 0.72). However, clinicians must balance this against the high rate of drug discontinuation seen in the trial (46%). Monitoring renal function (CrCl) is essential as dabigatran is 80% renally cleared, and the risk of bleeding—while not statistically significantly increased for 'major' bleeds in the trial—is higher when combined with antiplatelet therapy.

Fellow
Fellow

The MANAGE trial's primary composite endpoint included 'vascular death, MI, stroke, peripheral arterial embolism, amputation, and symptomatic VTE.' Which specific component showed the most robust benefit, and how does this affect the interpretation of dabigatran's role in 'cardiac' vs 'vascular' protection post-MINS?

Key Response

The reduction in stroke (HR 0.20) was one of the most striking findings in MANAGE, whereas the reduction in MI alone was not statistically significant. This suggests that dabigatran's primary benefit in MINS may be more related to preventing embolic or thrombotic stroke in a vulnerable population rather than solely preventing recurrent myocardial events, highlighting the broad 'vascular' risk associated with MINS.

Attending
Attending

Given the high rate of study drug discontinuation (46%) in the MANAGE trial, how does this 'intention-to-treat' reality affect your confidence in prescribing dabigatran for MINS in a real-world surgical population with polypharmacy and potential post-operative complications?

Key Response

High discontinuation rates often suggest poor tolerability or logistical hurdles (e.g., dyspepsia, bleeding concerns, or surgical re-interventions). While the ITT analysis remained significant, the real-world benefit may be lower if patients cannot remain on therapy for the intended duration. It teaches that while the evidence supports the drug, patient selection and adherence strategies are as critical as the prescription itself.

Scholarly Review

Critical appraisal through the lens of expert reviewers and guideline development

PhD
PhD

The MANAGE trial was terminated early due to slow recruitment and underwent a mid-trial modification of the primary composite endpoint. How do these factors potentially inflate the treatment effect, and what statistical adjustments are necessary to mitigate the 'winner's curse' in this context?

Key Response

Early termination for futility or slow recruitment, combined with endpoint modification (adding VTE and amputation), can lead to an overestimation of treatment effects (Type I error inflation). A PhD-level critique would look for sensitivity analyses using the original endpoint and evaluate the alpha-spending functions used to ensure that the reported p-values remain robust despite the trial's evolution.

Journal Editor
Journal Editor

The MANAGE trial utilized a definition of 'major bleeding' that may differ from the ISTH or TIMI criteria used in other NOAC trials. As a reviewer, how would you challenge the authors on the safety profile, specifically regarding the 'clinically important' but non-major bleeding events?

Key Response

Reviewers would note that while 'major bleeding' (life-threatening or critical organ) was not significantly higher, 'clinically important' bleeding was higher in the dabigatran group. Editors look for whether the 'net clinical benefit' accounts for minor bleeds that, in a post-surgical patient, can lead to wound complications, hematomas, and prolonged hospital stays, which are often not captured in a traditional 'major bleeding' metric.

Guideline Committee
Guideline Committee

The 2020 ESC and 2017 CCS guidelines emphasize the detection of MINS via troponin screening. Should MANAGE support a Class I recommendation for dabigatran in all MINS patients, or should it remain a lower-tier recommendation based on the evidence's limitations?

Key Response

Current guidelines generally give dabigatran a Class IIb recommendation for MINS because it is supported by only one large trial with significant discontinuation rates. Unlike the POISE-1 (Aspirin) or the use of statins, the evidence for dabigatran is promising but lacks the reproducibility across multiple large-scale RCTs required for a Class I 'must-do' recommendation in the perioperative standard of care.

Clinical Landscape

Noteworthy Related Trials

2008

POISE Trial

n = 8,351 · Lancet

Tested

Metoprolol succinate extended-release

Population

Patients undergoing non-cardiac surgery

Comparator

Placebo

Endpoint

Composite of cardiovascular death, nonfatal myocardial infarction, and nonfatal cardiac arrest

Key result: Metoprolol reduced the risk of myocardial infarction but increased the risk of death and stroke.
2009

RE-LY Trial

n = 18,113 · NEJM

Tested

Dabigatran etexilate 110 mg or 150 mg

Population

Patients with atrial fibrillation

Comparator

Warfarin

Endpoint

Composite of stroke or systemic embolism

Key result: Dabigatran 150 mg was superior to warfarin in preventing stroke, while 110 mg was non-inferior with lower major bleeding rates.
2014

POISE-2 Trial

n = 10,010 · NEJM

Tested

Clonidine or Aspirin

Population

Patients undergoing non-cardiac surgery at risk for cardiovascular complications

Comparator

Placebo

Endpoint

Composite of death and nonfatal myocardial infarction

Key result: Neither clonidine nor aspirin reduced the rate of the primary composite outcome in patients undergoing noncardiac surgery.

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